The combination of various MR techniques can make the course of Wallerian degeneration visualization .

联合运用多种MR成像技术研究 沃勒 变性，可使其病程 变化 变得可视化。

The application of various MRI techniques on Wallerian degeneration after cerebral infarction from imaging principle and pathogenesis were reviewed in this article .

本文从成像原理及发病机制等方面，对多种磁共振成像技术在脑梗死 沃勒 变性中的应用进行综述。

Objective : To study the repairing effects of peripheral nerve injury in Wallerian degeneration .

目的：研究 瓦勒 变性期修复神经损伤的效果。

The pathology of RFTC group was characterized by Wallerian degeneration and axon regeneration .

射频热凝组神经以 沃勒 变性和轴突再生为特点。

BACKGROUND : Ola rats is a kind of rats with genovariation who displays Wallerian degeneration after peripheral neuroaxonal damage that is slower than that normal 6J rats thereby additional damage factor may help fully understand the property of Ola rats .

背景：Ola鼠是一种基因变异鼠，其周围神经轴突损伤后 华勒氏 变性速度比正常的6J鼠缓慢，增加损伤因素可能有助于了解Ola鼠的特性。

Objective : To explore the value of the diffusion tensor imaging ( DTI ) in diagnosing Wallerian degeneration .

目的：探讨磁共振弥散张量成像技术（DiffusionTensorImaging，DTI）在 Wallerian 变性中的诊断价值。

Conclusion In Ola mice treated with ACR the Wallerian like degeneration delayed . However in 6J mice neurofilaments and mitochondria accumulation could be observed within axons .

结论Ola鼠对ACR 所致的类 华勒氏 变性 反应是延迟的，而6J鼠中毒后出现轴突肿胀、 变性，电镜下以神经微丝的聚集、线粒体堆积为特征；

Results : The course of Wallerian degeneration Schwann 's cell proliferation the thickness of myelin sheath the regeneration of axon and medullary sheath and the recovery of SFI and SNCV in ultrasound group were significantly earlier than those in control group ( P0.01 or P0.05 ) .

结果：超声组 损伤 神经 远 侧 Wallerian 变性 进程 加速、雪旺细胞增殖、变性组织吸收、轴索及髓鞘再生、感觉传导速度及坐骨神经功能的恢复等与对照组比较均提前（P0.01或P0.05）。

Results The test group showed Wallerian degeneration . The number of Schwann cell began to decrease at 24 hour surprisingly be rare at day 3 and 4 then began to increase from day 8 and formed Bungner belt after 14 days .

结果试验组显示 华勒氏 变性，雪旺细胞数在24小时后下降，3～4天后罕见，8天开始增加，14天形成Bungner带。

DTI would be an effective method to detect the state and time course of Wallerian degeneration in corticospinal tract after cerebral infarction .

DTI有利于检测脑梗死后皮质脊髓束 发生 Wallerian 变性的程度及 动态 演变过程。

Marked degeneration of myelin and Wallerian degeneration could be observed though without changes in the number of myelinated nerve fibers .

电镜下 见髓鞘明显变性即髓鞘 板 层 结构 破坏 和 瓦勒氏 变性，但有髓神经纤维计数无明显变化。

Results It showed that the electromagnetic field accelerated the course of Wallerian degeneration Schwann 's cell proliferation regeneration of axon myelin recovery of motor nerve conduction velosity of sciatic nerve .

结果电磁场 治疗加速了 损伤 神经 远 段 Wallerian 变性进程，促进雪旺氏细胞增殖，促进轴索及髓鞘再生， 加速运动神经传导速度的恢复。

Role of diffusion tensor imaging in diagnosing Wallerian degeneration

磁 共振弥散张量成像对 Wallerian 变性诊断价值的 评价

Then the nerve root underwent axons degeneration disruption and distal Wallerian degeneration while the MEP manifested the prolonged latency and the decreased amplitude .

继而脱髓鞘的神经轴突发生变性、断裂及远侧的 瓦勒氏 变性。MEP则表现为在潜伏期延长的同时伴有波幅降低，且MEP在 不同 程度的神经 病理 损伤 中 具有 相应 的 特征性表现形式。

Wallerian degeneration of the pyramidal tract after cerebral infarction studied by magnetic imaging

脑梗塞后锥体束 华 勒 变性的磁共振影像分析

MRI of Wallerian degeneration in central nervous system

沃勒 变性在中枢神经系统的MRI表现

Conclusions : DTI could detect Wallerian degeneration of CST as early as at the 3 days after infarction attack .

结论：DTI可以在脑梗死后早期检测到 皮质脊髓 束 的 Wallerian 变性，最早为发病后第3天。

Repairing effects of peripheral nerve injury in Wallerian degeneration

周围神经损伤的 瓦勒 变性 期修复

A morphological study on the occurrence of wallerian degeneration of rat sciatic nerves after nerve cutting and preventive effect of gangliosides

神经节苷脂预防 华勒 变性的显微结构研究

Microscopic examination showed that the myelinated nerve fibers exhibited Wallerian degeneration and axon regeneration and the cytochondria in cylindraxile were severely injured or disappeared in group RFTC .

与N组比较，RFTC组术后有髓神经纤维的形态学以 沃勒 变性和再生为特点，轴索内线粒体严重损伤或消失；

Objective To review the pathologic changes of Wallerian degeneration ( WD ) and secondary transneuronal degeneration ( TD ) and evaluate their MRI in central nervous system .

目的回顾 沃勒 变性及继发跨神经元退变的病理学改变，分析其在中枢神经系统的MRI表现。

In recent years there were many experimental reports about pathophysiological process of Wallerian degeneration after cerebral infarction but these studies lacked visibility .

近年来许多 学者 通过 动物实验研究脑梗死后 沃勒 变性的病理生理过程，但缺乏可视性。

Conclusions : Adult rat SC undergoing Wallerian degeneration can still synthesize and secrete neurotrophic factors under serum free cultivation the activities of which existed in > 10kD fraction .

结论：成年大鼠 瓦勒 变性期SC在无血清培养条件下仍能合成与分泌神经营养因子，其活性存在于＞10kD蛋白分子中。