Pathogenesis were due to D-Galn substitute for uridine diphosphoglucose ( UDPG ) way of metabolism .

病变机理是过量D－ Galn入肝代替了尿苷二磷酸葡萄糖的代谢途径所引起的。

Among the proteins identified the immobilized cultures enhanced the expressions of UDPG pyrophosphorylase which was involved directly or indirectly in the biosynthesis of cell wall polysaccharides .

固定化培养增强了细胞中 UDPG pyrophosphorylase的表达，该酶直接或间接参与细胞壁多糖的合成。

UDPG PPlase activity was very low in the early period of growing seed and quickly became high during the mature to the beginning of storage and kept in high lever for long time .

银杏种子幼小至膨大期， UDPGPPlase活性较低；种子成熟期至贮藏初期，活性迅速而显著地增强；