In high-frequency tetanic contraction the maximal tension ( Po ) of soleus was reduced respectively by 32.8 % 60.1 % and 77.6 % after one ( P < 0.05 ) two and four weeks of ( P < 0.01 ) tail-suspension ( SUS ) .

与对照组比较，悬吊1wk大鼠SOL高频 强直 收缩的最大张力（Po）降低32.8%，但统计学无差异（P>0.05）；悬吊2wk组降低60.1%（P<0.01）；

P_ 33 / P_o ( P_ 33 transient tension of tetanic contraction at the 33 s ) was reduced significantly in SUS and it manifested an increase in fatigability during tetanic contraction in SUS .

但是，高频 强直 收缩第33秒 收缩张力与最大张力之比（P33/Po）却明显降低。

Objective To elucidate the underlying mechanisms of the high fatigability in mouse atrophic soleus during high-frequency tetanic contraction .

目的探讨萎缩比目鱼肌高频 强直 收缩疲劳性增加涉及细胞膜离子通道的可能机理。

No influence of increased frequency on fatigability of tetanic contraction in rat atrophic soleus

增加刺激频率不影响大鼠萎缩比目鱼 肌强直 收缩疲劳性

Conclusion These results suggest that the changes in K + channel characteristics may increase the fatigability during high-frequency tetanic contraction in atrophic soleus .

结论萎缩比目鱼肌纤维膜上K+通道的改变，可能是引起其高频 强直 收缩疲劳性增加的主要因素。