vacuolar degeneration

空泡变性

  • Pathologically hepatocytes in high Mo groups showed granular degeneration and vacuolar degeneration .

    病理形态学 观察,高钼组 雏鸡肝细胞呈现不同 程度的颗粒变性和 空泡 变性

  • After 24 hours hepatic cells swelled and occurred vacuolar degeneration and punctiform necrosis .

    保存24h后,肝细胞出现肿胀、 空泡 变性及点状坏死,肝窦内皮 细胞肿胀、 变性,并开始向窦腔内脱落。

  • Histopathology transformations : Liver vacuolar degeneration and swollen in hepatocytes small bile ducts proliferated .

    组织病理学变化:肝脏,肝细胞 空泡 变性,胆管增生。肝小叶内和 管区小灶 细胞浸润和 酸性 细胞浸润,及后期肝细胞 极度肿胀;

  • Capillary basement membrane thickened locally foot processes fusion of visceral epithelial cells were found in varying degrees accompanied by vacuolar degeneration of some mitochondrial .

    毛细血管基底膜局部增厚, 肾小球脏层上皮细胞出现不同程度的足突融合 现象并伴有部分线粒体 空泡 变性

  • Histopathology showed hyperkeratosis accompanied by focal parakeratosis vacuolar degeneration of basal layer and superficial dermal bandlike infiltration composed of lymphocytes and also eosinophil and plasma cells .

    组织病理改变主要为角化过度伴灶状角化不全,基底层 液化 变性,真皮浅层可见淋巴细胞大致呈带状分布,并可见嗜酸性粒细胞和浆细胞浸润。

  • Results The adhesion disturbance lodge and squamous metaplasia of cilia in tracheal epithelium ; and vacuolar degeneration in epithelium cells were observed .

    结果气管上皮纤毛粘连、乱、伏及鳞状化生,上皮细胞 空泡 变性,并 可见 新生的纤毛上皮细胞。

  • The hepatic cells and the epithelial cells of renal tubules were vacuolar degeneration tracheal mucosa epithelial layer arterial hyperemia and hemorrhage .

    组织 病变 表现 神经 空泡 肺泡内出血和水肿,肝细胞与肾小管上皮细胞明显 水泡 变性,气管粘膜上皮层下高度充血、出血。

  • The liver showed edema vacuolar degeneration and foci necrosis .

    肝水肿, 间隙 ,肝细胞 空泡 变性及坏死;

  • The renal tubules show varying degrees of vacuolar degeneration necrosis and cast formation .

    肾小管呈现 空泡 、坏死及管型形成。

  • There were vacuolar degeneration in renal tubular cells and the tubular lumens were narrowed .

    肾小管出现细胞 空泡 变性,管腔变窄。

  • Results In the infected model group there were obvious swelling karyopyknosis and destruction of the structure in the brain cells as well as myelin sheath solution and vacuolar degeneration in the mitochondrion and crest were destroyed .

    结果电镜下感染模型组小鼠脑细胞胞浆水肿明显.核仁固缩,核内结构破坏,多数线粒体 空泡 改变,核仁内 可见 病毒 颗粒内脊破坏、髓鞘严重松解、破坏。

  • The renal tubular epithelial cells were vacuolar degeneration necrosis . There were cell debris and tube-type in the lumen of renal tubular .

    M组 肾小球变形皱缩,肾小管上皮细胞 空泡 变性细胞片状坏死,肾小管管腔变窄,其内出现细胞碎片或管型形成,肾间质可见出血和炎性细胞浸润。

  • Under an electron microscope it was observed that the cytoplasm of KC in the GC + DENA group contained a large number of electron dense granules or KC showed vacuolar degeneration or necrosis ;

    电镜下证实:GC+DENA组肝窦内枯否细胞(KC)胞浆内含有大量电子致密物或细胞 空泡 变性及坏死;

  • Vacuolar degeneration of axon and ICC-like cells was revealed by electron microscopy in myenteric plexus of cathartic colon .

    电镜下见肌间丛 神经轴突空化,ICC样细胞 变性

  • Jiawei Yinchenhao Decoction low dose group of rat liver tissue structure failure liver cell degeneration necrosis inflammatory cell run lighter than in the model group showed moderate diffuse vacuolar degeneration .

    加味茵陈蒿汤低剂量组大鼠肝组织结构部分破坏,肝细胞变性、溶解坏死、炎性细胞润较模型组轻,呈中度弥漫性 空泡

  • In group DM glomerular basement membrane thickening a large number of mesangium matrix deposition vacuolar degeneration of tubular epithelial cells renal interstitial fibrosis and mononuclear cells and lymphoid cell infiltration appears .

    糖尿病组大鼠肾小球毛细血管基底膜增厚,系膜区基质大量沉积,肾小管上皮细胞 空泡 变性,肾间质出现纤维化及单核细胞和淋巴样细胞浸润。

  • Pathologic changes : In group A the tissue structure is clear without edema vacuolar degeneration cloudy swelling and necrosis .

    病理观察:A组各组织结构清晰,未见水肿、 空泡 变性、浊肿及坏死。

  • Vacuolar degeneration in the renal tubular epithelium and the liver cells ;

    肾小管上皮细胞 空泡 变性肾小球萎缩;

  • The mitochondrium in the ischemia / reperfusion model group appeared vacuolar degeneration .

    脑缺血再灌注模型组线粒体呈 空泡

  • Vacuolar degeneration was checked in brain heart kidney spleen and ballocks or ovaries .

    病理组织学观察可见脑、心脏、肾、脾、睾丸和卵巢等组织发生明显 空泡 变性

  • The results showed that : ① major pathological changes was fatty and vacuolar degeneration of hepatocytes cloudy swelling of renal epithelial cells and myocardial cells ;

    结果表明:① 青霉 中毒 尼西 的主要病理变化是肝细胞脂肪 变性、肾小管上皮细胞浊肿、心肌细胞 颗粒 变性

  • Epidermal changes consist of acanthosis vacuolar degeneration parakeratosis hyperkeratosis and necrosis .

    上皮的变化包括棘细胞增生, 空泡 变性,不全角化,过度角化和坏死等。

  • At histopathological level the hepatocytes epithelial cells of renal tubule and cardiac muscle cells mainly showed granular and vacuolar degeneration .

    肝细胞、肾小管上皮细胞、心肌细胞颗粒变性和 空泡 变性

  • The skin showed eosinophilic necrosis of epidermal cells vacuolar degeneration of epidermal basal cells epidermal cleft bulla and lymphocytic infiltration of the dermis .

    皮肤表现为表皮细胞嗜酸性坏死和基底层细胞 空泡 ,表皮发生裂隙、大泡形成以及真皮淋巴细胞浸润。

  • Obvious liver structure of rats in model group hepatic lobule structure was not clear liver cells arranged in disorder liver cell degeneration necrosis hepatic periportal inflammatory cell infiltration liver cell diffuse vacuolar degeneration .

    模型组大鼠肝脏结构破坏较明显,肝小叶结构欠清,肝细胞索排列紊乱,肝细胞变性、溶解坏死、肝汇管区炎性细胞浸润明显,肝细胞呈弥漫性 空泡

  • Pathologic examination revealed that hippocampal neurons displayed local ischemic damage cells in granular layer showed severe edema degeneration as well as necrosis and neuronal mitochondria showed vacuolar degeneration .

    病理形态学显示神经元呈局部缺血性改变,颗粒层细胞高度水肿、变性、坏死,神经元线粒体 空泡 现象